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Klonopin contains the ingredient, Clonazepam, which is approved for use as treatment for anxiety by the U.S. Food and Drug Administration. It has been proven that Klonopin reduces the symptoms of anxiety. Anxiety can have its roots in a feeling of nervousness of an upcoming event. If we are unable to contain the nervousness, then it advances to fear. If we are unable to go about our daily routine because of fear of this upcoming event, then the condition has blossomed into an anxiety disorder.

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Klonopin

Klonopin (Clonazepam) is a benzodiazepine derivative with anticonvulsant, muscle relaxant, and anxiolytic properties. It is marketed by Roche under the trade-names Klonopin in the United States, and Ravotril in Chile. Other names like Rivotril or Rivatril are known throughout the large majority of the rest of the world. Clonazepam is a chlorinated derivative of nitrazepam and therefore a nitrobenzodiazepine.

DRUG CLASS AND MECHANISM: Klonopin no prescription is an anti-anxiety medication in the benzodiazepine family, the same family that includes diazepam (Valium),  alprazolam (Xanax), lorazepam (Ativan), flurazepam (Dalmane), and others. Klonopin no prescription and other benzodiazepines act by enhancing the effects of gamma-aminobutyric acid (GABA) in the brain. GABA is a neurotransmitter (a chemical that nerve cells use to communicate with each other) that inhibits brain activity. It is believed that excessive activity in the brain may lead to anxiety or other psychiatric disorders. Klonopin no prescription also is used to prevent certain types of seizures.

PRESCRIPTION: Not required at our pharmacy. You will be given a prescription online.

GENERIC AVAILABLE: Yes

PREPARATIONS: Tablets: 0.5, 1, and 2 mg. Disintegrating tablets: 0.125, 0.25, 0.5, 1, and 2 mg.

STORAGE: Tablets should be kept at room temperature, between 15° and 30°C (59-86°F).

PRESCRIBED FOR: Klonopin no prescription is used for the short-term relief of symptoms of anxiety. It is used for certain types of seizures, specifically petit mal seizures, akinetic seizures, and myoclonus, as well as Lennox-Gastaut syndrome. Klonopin no prescription maybe used alone or together with other medications for these seizure disorders.

DOSING: The dose of Klonopin no prescription is tailored to the patient’s needs. For seizures in adults the initial dose is 1.5 mg daily in 3 divided doses. Dosage may be increased by 0.5 to 1 mg daily every 3 days until seizures are controlled or side effects preclude further increases in dose. The maximum dose is 20 mg daily. The initial dose for panic disorders is 0.25 mg twice daily. The dose may be increased to the target dose of 1 mg daily after 3 days.

DRUG INTERACTIONS: Klonopin no prescription, like all other benzodiazepines, accentuates the effects of other drugs that slow the brain’s processes, such as alcohol, barbiturates, and narcotics and leads to increased sedation.

PREGNANCY: Klonopin no prescription and other benzodiazepines have been associated with fetal damage, including congenital malformations, when taken by pregnant women in their first trimester. Klonopin no prescription is best avoided in the first trimester and probably throughout pregnancy.

NURSING MOTHERS: Benzodiazepines are secreted in breast milk. Mothers who are breastfeeding should not take Klonopin no prescription.

SIDE EFFECTS: The most commonly noted side effects associated with Klonopin no prescription are sedation, which is reported in approximately half of patients, dizziness which is reported in one-third of patients, weakness, and unsteadiness. Other side effects include a feeling of depression, loss of orientation, headache, and sleep disturbance.

Like all benzodiazepines, Klonopin no prescription can cause physical dependence. Suddenly stopping therapy after a few months of daily therapy may be associated with a feeling of loss of self-worth, agitation, and insomnia. If Klonopin no prescription is taken continuously for longer than a few months, stopping therapy suddenly may produce seizures, tremors, muscle cramping, vomiting, and/or sweating. Therefore, discontinuation usually is accomplished by reducing the dose gradually.

Antiepileptic medications have been associated with an increased risk of suicidal thinking and behavior.  Anyone considering the use of antiepileptic drugs must balance this risk of suicide with the clinical need for the antiepileptic drug.  Patients who begin antiepileptic therapy should be closely observed for clinical worsening, suicidal thoughts or unusual changes in behavior.

Klonopin Pharmacodynamics

The precise mechanism by which klonopin no prescription exerts its antiseizure and antipanic effects is unknown, although it is believed to be related to its ability to enhance the activity of gamma aminobutyric acid (GABA), the major inhibitory neurotransmitter in the central nervous system. Convulsions produced in rodents by pentylenetetrazol or, to a lesser extent, electrical stimulation are antagonized, as are convulsions produced by photic stimulation in susceptible baboons. A taming effect in aggressive primates, muscle weakness and hypnosis are also produced. In humans, klonopin no prescription is capable of suppressing the spike and wave discharge in absence seizures (petit mal) and decreasing the frequency, amplitude, duration and spread of discharge in minor motor seizures.

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klonopin no prescription is rapidly and completely absorbed after oral administration. The absolute bioavailability of klonopin no prescription is about 90%. Maximum plasma concentrations of klonopin no prescription are reached within 1 to 4 hours after oral administration. klonopin no prescription is approximately 85% bound to plasma proteins. klonopin no prescription is highly metabolized, with less than 2% unchanged klonopin no prescription being excreted in the urine. Biotransformation occurs mainly by reduction of the 7-nitro group to the 4-amino derivative. This derivative can be acetylated, hydroxylated and glucuronidated. Cytochrome P-450 including CYP3A, may play an important role in klonopin no prescription reduction and oxidation. The elimination half-life of klonopin no prescription is typically 30 to 40 hours. klonopin no prescription pharmacokinetics are dose-independent throughout the dosing range. There is no evidence that klonopin no prescription induces its own metabolism or that of other drugs in humans.